Abnormal accumulation of amyloid proteins in the central nervous system (CNS) results in various neurological disorders. Alzheimer's disease (AD), a prevalent disorder, is characterized by amyloid plaque buildup in the brain, primarily beta-amyloid proteins. These plaques impede neuronal communication and cause neurodegeneration, a hallmark of AD. Similarly, cerebral amyloid angiopathy (CAA) involves amyloid deposits impacting brain blood vessels, elevating stroke and hemorrhage risk. Consequently, both Alzheimer's disease and cerebral amyloid angiopathy stem from amyloid protein accumulation within the CNS.