Step 1: Link each necrosis pattern to its usual setting. Coagulative goes with ischemia in heart, kidney, and spleen; liquefactive goes with brain infarcts and pus; fibrinoid goes with antigen-antibody complex damage to vessels. Step 2: In immune vasculitis, complexes plus fibrin accumulate within the arterial or arteriolar wall, giving a glassy eosinophilic deposit termed fibrinoid necrosis. Step 3: This same change marks the vessels in malignant hypertension, reinforcing the vascular and immune association. Step 4: Because the stem specifies blood vessels and immune reactions, fibrinoid necrosis is the only fitting choice. \[\boxed{\text{Fibrinoid}}\]