Step 1: Start from the central problem in hypovolemic shock - the tank is empty, so venous return, stroke volume, and cardiac output all drop.
Step 2: Baroreceptor unloading triggers a massive sympathetic surge plus activation of renin-angiotensin. The aim is to defend perfusion of the brain and heart by clamping down everywhere else, raising systemic vascular resistance.
Step 3: Within the renal circulation this intense catecholamine drive constricts the afferent arteriole, cutting renal blood flow and protecting the rest of the circulation - the limbs go cold and shut down.
Step 4: Rule out the alternatives: renal flow goes down not up, cardiac output is depressed rather than normal, and isolated efferent constriction is the GFR-sparing mechanism of moderate hypovolemia, not the hallmark of established shock. \[\boxed{\text{Afferent arteriolar constriction}}\]