Picture Guillain Barre as an autoimmune assault on the peripheral nerves and roots, usually a week or two after a gut or respiratory infection. Three of the four options are textbook truths, and the trick is to find the one that does not belong to a peripheral process.
The weakness famously creeps upward from the feet, so ascending paralysis is correct. Since the damage is at the peripheral nerve, a lower motor neuron level, the muscles go limp and the reflexes vanish, giving flaccid paralysis, also correct. The cerebrospinal fluid carries the signature of high protein with almost no extra cells, the $albuminocytological\ dissociation$, again correct.
The outlier is a sensory level. A crisp band below which all sensation is lost is the language of a spinal cord lesion, a myelopathy, not a neuropathy. GBS may cause patchy sensory symptoms, but it never draws a sharp sensory level across the trunk. That statement is therefore the false one being asked for.
So the exception is the sensory level.
\[\boxed{\text{Sensory level}}\]