Step 1: Frame this around the mechanism - ethanol and its metabolites poison cardiac myocytes over years of heavy drinking. Step 2: The weakened muscle can no longer contract forcefully, so the chambers stretch out and the heart becomes large and floppy with a reduced ejection fraction. Step 3: A large, poorly contracting, stretched heart is the definition of the dilated pattern, which places alcoholic cardiomyopathy under dilated cardiomyopathy. Step 4: The thick-walled hypertrophic type is hereditary, and the two -itis options describe inflammation rather than the alcohol-driven dilation, so they are excluded. \[\boxed{\text{Dilated cardiomyopathy}}\]